Difference between revisions of "AssociativeMemoryResearch"

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Associative Memory Research

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Associative Memory (AM) research covers technologies enabling implementation of associative memory which enables thought process and links previous experience to novel situations.

Technologies

• Kohonen networks

Types of Associations

Feel the difference between:

• Clear concept can be restored from noisy data
• Most related concept can be restored by its small part
• Several concepts can be derived from feature/another concept

Thoughts

• Pribram's model*

• alternative to the transcortical model of neocortical organization

* extrinsic sectors (primary projection areas) - neocortical areas whose fibers enter or leave the cerebral hemispheres
* intrinsic sectors (association areas) - their fibers remain within the cerebrum

• principal interaction of extrinsic and intrinsic systems occurs at the thalamic level

* contribution of intrinsic neocortex to the final output of the extrinsic system is mediated by the convergence of influences from both intrinsic and extrinsic systems by subcortical mechanisms
* intrinsic system may influence also the input of the extrinsic systems by regulation of peripheral sensory mechanisms

Interesting Pictures

• Human Memory Systems - see [[1]]

• Cognitive Cycle - see [[2]]

• Generic Auto-Associative Memory - see [[3]]

• Context Binding - see [[4]]

Articles Review

Multi-Associative Memory in fLIF Cell Assemblies (CA)

see [[5]].

• Based on:*
• Hebbs Cell Assembly Theory (CA is neural basis for concepts)
• network of biologically plausible fLIF (fatiguing, Leaky, Integrate and Fire) neurons

• Introduction, Background:*
• hypo: Concepts are stored as CAs, associations are connections between CAs
• concepts connected as 1-1,1-N,N-M
• associations can be context-sensitive - retrieval of an associated concept can be based on a combination of the base concept and the context
• AM features: priming, differential associations, timing, gradual learning and change, encoding instances (and others)

• CAs and auto-associative memory*:
• CA theory: objects, ideas, stimuli and even abstract concepts are represented in the brain by simultaneous activation of large groups of neurons with high mutual synaptic strengths
• *long-term memory*: neurons are learned by Hebbian rule from mutual activation, gradually assembling into CAs after repeated and persistent activation
• *short-term memory:* CA is activated when its certain number of neurons is activated, then CA reverberates due to high mutual synaptic strengths
• CA is a form of auto-associative memory
• *Hopfield Model*: binary neurons, well-connected network, bidirectional weighted connections, Hebbian learning

• CAs and multi-associative memory*:
• Psychologically, memories are not stored as individual concepts, but large collections of associated concepts that have many to many connections
• repeated co-activation of multiple CAs result in the formation of multiple and sequential associations, and sometimes new CAs

• Multi-associative memory models*:
• *Non-Holographic Associative Memory* (1969): well-connected network that can learn to map input bit patterns to output bit patterns; input CAs are connected to output CAs via learned one way associations
• *The Linear Associator* (Kohonen, 1977): feed-forward, well connected network;
• *Multi Modular Associative Memory* (1999): well connected modules, resilient to corrupted input
• *Valiant model* (2005): random graphs, biologically implausible learning, theoretical model of memorisation and association based on four quantitative parameters associated with the cortex:

* the number of neurons per concept
* number of synapses per neuron
* synaptic strengths
* number of neurons in total

• *Interactive activation model* (1981): each concept is represented by a node, and connections are made between nodes to show how closely related these are; not well connected
• Finally:

* simulated neural systems can encode multi-associative memories
* well connected systems are not a good model of the brain
* use partitioning the system into modules, and sparsely connected random graphs
* there models do not account for some human characteristics, e.g. context effects

• Computation model for simulation*:

• fLIF neural network:

- fLIF neurons collect activation from pre-synaptic neurons and fire on surpassing a threshold T
- on firing, a neuron loses its activation level, otherwise the activation leaks gradually:
Ait = Ait-1/d + Sum( Wij * Sj ).
d - decay factor.
- firing is a binary event, and activation of Wij is sent to all neurons j to which the firing neuron i has a connection.
- fatiguing causes the threshold to be dynamic:
t+1 = Tt + Ft.
- Ft is positive (F+) if the neuron fires at t and negative (F-) otherwise

• Network architecture:

- network is a whole or split into several subnetworks (for some simulations)
- intra-subnet synapses are biologically inspired distance biased connections (most likely excitatory connections to neighbouring neurons)
- subnet is a rectangular array of neurons with distance organized toroidally
- inhibitory connections within a subnet and all inter-subnet connections are set up randomly
- connectivity rule for excitatory neurons; connection i->j exists if Cij=1:
Cij = 1; if r < 1/(d*v)
r - random between 0 and 1
d - the neuronal distance (value=5 works well for all simulations)
v - the connection probability
- long distance intra-network connections are inspired by biological long distance axons with many synapses
- networks are divided into multiple CAs in response to stimuli using unsupervised learning algorithms
- the CAs are orthogonal and represent different concepts, and this is based on training